clm-13a6rtv5w7

BLA (Basolateral amygdalar nucleus) functionally connects to CEA (Central amygdalar nucleus)

All claims for this connection

Species: Mouse
Result: present
Method: electrical stimulation
Sign: excitatory
Evidence: Electrical stimulation with an electrode placed in the ventral BLA close to the CeA border evoked short, stable-latency excitatory postsynaptic currents in CeC/L neurons held at -70 mV in brain slices from naive, saline- and formalin-treated mice. Pregabalin reduced these BLA-evoked EPSCs and raised the paired-pulse ratio only in formalin-treated animals, indicating a presynaptic site of action at BLA terminals; EPSC amplitude at fixed 100 µA stimulation did not differ between saline and formalin groups.
Source: Input-dependent synaptic suppression by pregabalin in the central amygdala in male mice with inflammatory pain
Neurobiology of Pain, 2021
DOI 10.1016/j.ynpai.2021.100078 · PubMed 34877437 · PMC8628014, Figs. 1A, 2, 3, 5; Results
Licence: CC-BY-4.0
Named in the paper: “BLA (basolateral amygdala)” and “CeC/L (capsular and lateral part of the central nucleus of the amygdala)”
Made by: an AI model reading the paper (claude-opus-5, extract@0.3.0), 2026-10-09
Checked by: a second AI model (claude-opus-5-5) agrees, 2026-10-09
“Stimulating the ventral BLA evoked EPSCs in CeC/L neurons in mouse slices; the paper also reports the pregabalin and paired-pulse effects only in formalin mice and no saline–formalin amplitude difference at 100 µA.”
Status: proposed
Drafted and checked by AI models; it stays proposed until people have audited a sample of such claims.