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BLA (basal amygdaloid nucleus) functionally connects to CEA (central amygdaloid nucleus)

All claims for this connection
Species
Rat
Result
present
Method
electrical stimulation
Sign
inhibitory
Evidence
BLA stimulation also evoked longer, more variable-latency IPSCs in CeLC neurons that failed to follow high-frequency stimulation. These IPSCs were blocked by bicuculline and reduced by NBQX, consistent with glutamate-driven disynaptic feedforward inhibition, and they were reduced in arthritic rats.
Source

Pain-Related Increase of Excitatory Transmission and Decrease of Inhibitory Transmission in the Central Nucleus of the Amygdala are Mediated by mGluR1

Molecular Pain, 2010

DOI 10.1186/1744-8069-6-93 · PubMed 21162731 · PMC3016348, Fig. 1C-H; Fig. 2B; Fig. 3
Named in the paper
“BLA” and “CeLC (latero-capsular division of the CeA)”
Made by
an AI model reading the paper (claude-opus-5-5, extract@0.3.0), 2026-10-09
Checked by

a second AI model (claude-opus-5-5) agrees, 2026-10-09

“Fig. 1–3 show rat BLA stimulation evoking polysynaptic, bicuculline-blocked, NBQX-sensitive IPSCs in CeLC that were reduced in arthritis.”

Status

proposed

Drafted and checked by AI models; it stays proposed until people have audited a sample of such claims.