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TH (Thalamus) functionally connects to Rat basal amygdaloid nucleus principal neurons

Species: Rat
Result: present
Method: electrical stimulation
Sign: excitatory
Evidence: Electrical stimulation of thalamic afferent fibers in amygdala slices evoked monosynaptic EPSCs in BLA principal neurons; the NMDA receptor component was isolated with CNQX and picrotoxin at +40 mV and progressively blocked by MK-801 during repeated stimulation, with a faster decay constant in slices from stressed rats (control 33.47 ± 2.3, n = 12; stress 25.03 ± 2.03, n = 12), indicating higher presynaptic release probability at this thalamic input ten days after acute stress.
Source: The delayed strengthening of synaptic connectivity in the amygdala depends on NMDA receptor activation during acute stress
Physiological Reports, 2016
DOI 10.14814/phy2.13002 · PubMed 27798355 · PMC5099964, Materials and Methods (electrophysiology); Fig. 2A–C
Licence: CC-BY-4.0
Named in the paper: “thalamic inputs (thalamic afferent fibers)” and “BLA principal neurons”
Made by: an AI model reading the paper (claude-opus-5, extract@0.3.0), 2026-10-09
Checked by: a second AI model (claude-opus-5) agrees, 2026-10-09
“The paper evokes monosynaptic glutamatergic EPSCs in BLA principal neurons by stimulating thalamic afferents, with the reported decay constants and sample sizes in rats.”
Status: proposed
Drafted and checked by AI models; it stays proposed until people have audited a sample of such claims.