clm-d86p69k57a
DR (Dorsal nucleus raphe) functionally connects to BLA glutamatergic principal neurons
All claims for this connection- Species
- Mouse
- Result
- absent
- Method
- optogenetic circuit mapping
- Sign
- unknown
- Evidence
- Unlike interneurons, basal amygdala principal neurons showed no fast glutamatergic EPSC to single light pulses activating raphe axons, indicating that glutamate-releasing 5-HT terminals do not contact these cells.
- Source
Control of Amygdala Circuits by 5-HT Neurons via 5-HT and Glutamate Cotransmission
The Journal of Neuroscience, 2017
DOI 10.1523/jneurosci.2238-16.2016 · PubMed 28087766 · PMC5320609, Results, 'Activation of 5-HT neurons evokes 5-HT-mediated responses in BA PNs'; Fig. 5Licence: CC-BY-NC-SA-4.0
- Named in the paper
- “DRN (5-HT neurons)” and “BA principal neurons (PNs)”
- Made by
- an AI model reading the paper (claude-opus-5, extract@0.3.0), 2026-10-10
- Checked by
a second AI model (claude-opus-5) agrees, 2026-10-10
“Paper explicitly reports no fast EPSCs in PNs (n=18) to single light pulses.”
- Status
proposed
Drafted and checked by AI models; it stays proposed until people have audited a sample of such claims.