clm-hcep52rhkr
BLA (Basolateral amygdalar nucleus) functionally connects to LA glutamatergic principal neurons
All claims for this connection- Species
- Mouse
- Result
- present
- Method
- electrical stimulation
- Sign
- unknown
- Evidence
- Electrical stimulation of the BL in slices evoked fixed-latency antidromic spikes in about a fifth of LA principal neurons, plus variable-latency polysynaptic IPSCs that were largely abolished by glutamate receptor blockade, consistent with recurrent recruitment of local LA interneurons.
- Measurements
- connection probability: 0.227, n = 22
- Source
Low-threshold spiking interneurons perform feedback inhibition in the lateral amygdala
Brain Structure and Function, 2020
DOI 10.1007/s00429-020-02051-4 · PubMed 32144495 · PMC7166205, Fig. 1a3, Fig. 1b; Results, 'BL Stimulation and inhibition in LA neurons'Licence: CC-BY-4.0
- Named in the paper
- “basolateral amygdala (BL)” and “LA principal neurons”
- Made by
- an AI model reading the paper (claude-opus-5-5, extract@0.3.0), 2026-10-09
- Checked by
a second AI model (claude-opus-5-5) disagrees, 2026-10-09
“The direction is reversed: the spikes were antidromic in LA axons projecting to BL, and the paper states LA-to-BL transfer is largely unidirectional, so BL is not shown to input LA principal cells.”
- Status
proposed
Drafted and checked by AI models; it stays proposed until people have audited a sample of such claims.