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BLA (basal amygdaloid nucleus) functionally connects to CEA (central amygdaloid nucleus)

Species: Rat
Result: present
Method: electrical stimulation
Sign: excitatory
Evidence: In rat slices, electrical stimulation in the BLA evoked short, fixed-latency EPSCs in CeLC neurons that followed 20 Hz stimulation and were abolished by the non-NMDA receptor antagonist NBQX. This indicates a monosynaptic glutamatergic input, and its input-output function was enhanced in arthritic rats.
Source: Pain-Related Increase of Excitatory Transmission and Decrease of Inhibitory Transmission in the Central Nucleus of the Amygdala are Mediated by mGluR1
Molecular Pain, 2010
DOI 10.1186/1744-8069-6-93 · PubMed 21162731 · PMC3016348, Fig. 1C-H; Fig. 2A
Named in the paper: “BLA” and “CeLC (latero-capsular division of the CeA)”
Made by: an AI model reading the paper (claude-opus-5-5, extract@0.3.0), 2026-10-09
Checked by: a second AI model (claude-opus-5-5) agrees, 2026-10-09
“Fig. 1–2A show rat BLA stimulation evoking fixed-latency, NBQX-blocked EPSCs in CeLC that follow 20 Hz, with I/O enhanced in arthritis.”
Status: proposed
Drafted and checked by AI models; it stays proposed until people have audited a sample of such claims.