Mouse
BLA (Basolateral amygdalar nucleus) functionally connects to BLA glutamatergic principal neurons
3 claims from 3 sources: 3 found, 0 tested and absent, 0 ambiguous. Evidence: electrical stimulation, optogenetic circuit mapping.
The claims behind it
Electrical stimulation within the BLA evoked mixed AMPAR/NMDAR excitatory postsynaptic currents in BLA pyramidal neurons, used as a measure of non-pathway-specific local input; the AMPA/NMDA ratio of this input was unchanged by stress.
DOI 10.1038/s41398-020-0837-3 · PubMed 32424318 · PMC7235080, Supplementary Fig. 1B,D,E (described in Results)Made by an AI model reading the paper (claude-opus-5, extract@0.3.0); a second AI model (claude-opus-5-5) agrees.
Local electrical stimulation under NBQX/APV evoked monosynaptic IPSPs and IPSCs in BA principal neurons. Baclofen abolished these responses, and their paired-pulse ratio depended on the inter-stimulus interval.
DOI 10.1016/j.neuron.2015.08.024 · PubMed 26402610 · PMC4590554, Fig. 7A-CMade by an AI model reading the paper (claude-opus-5-5, extract@0.3.0); a second AI model (claude-opus-5) agrees.
Sequential patterned-light activation of individual soma-targeted ChR2-expressing neurons within the basal amygdala, with glutamate receptors blocked by NBQX and L-APV, evoked IPSCs in non-expressing BA principal neurons, demonstrating local inhibitory connections; connection probability and quantal IPSC amplitude were both lower in NRXN1alpha knockouts (about 69-71 presynaptic cells probed per recorded neuron).
DOI 10.1038/s41398-020-00926-y · PubMed 32684634 · PMC7370229, Fig. 4a–fMade by an AI model reading the paper (claude-opus-5, extract@0.3.0); a second AI model (claude-opus-5) agrees.